Recent publications

1. Rossow, C.F., et al., NFATc3-Induced Reductions in Voltage-Gated K+ Currents After Myocardial Infarction. Circ Res, 2004. 94(10): p. 1340-50.Pubmed

2. Amberg, G.C., et al., NFATc3 regulates Kv2.1 expression in arterial smooth muscle. J Biol Chem, 2004.Pubmed

3. Amberg, G.C. and L.F. Santana, Downregulation of the BK channel β1 subunit in genetic hypertension. Circ Res, 2003. 93(10): p. 965-71.Pubmed

4. Amberg, G.C., et al., Modulation of the molecular composition of large conductance, calcium activated potassium channels in vascular smooth muscle during hypertension. J Clin Invest, 2003. 112(5): p. 717-24.Pubmed

5. Santana, L.F., et al., Functional coupling of calcineurin and protein kinase A in mouse ventricular myocytes. J Physiol, 2002. 544(Pt 1): p. 57-69.Pubmed

6. Heppner, T.J., et al., Alkaline pH shifts calcium sparks to calcium waves in smooth muscle cells of pressurized cerebral arteries. Am J Physiol Heart Circ Physiol, 2002. 283(6): p. H2169-76.Pubmed

7. Guatimosim, S., et al., Local Ca2+ Signaling and EC Coupling in Heart: calcium Sparks and the Regulation of the [Ca2+]i Transient. J Mol Cell Cardiol, 2002. 34(8): p. 941.Pubmed

8. Wellman, G.C., et al., Role of phospholamban in the modulation of arterial calcium sparks and calcium-activated potassium channels by cAMP. Am J Physiol Cell Physiol, 2001. 281(3): p. C1029-37.Pubmed

9. Ufret-Vincenty, C.A., et al., Role of sodium channel deglycosylation in the genesis of cardiac arrhythmias in heart failure. J Biol Chem, 2001. 276(30): p. 28197-203.Pubmed

10. Ufret-Vincenty, C.A., D.J. Baro, and L.F. Santana, Differential contribution of sialic acid to the function of repolarizing K+ currents in ventricular myocytes. Am J Physiol Cell Physiol, 2001. 281(2): p. C464-74.Pubmed

11. Santiago, J., et al., Probing the effects of membrane cholesterol in the Torpedo californica acetylcholine receptor and the novel lipid-exposed mutation alpha C418W in Xenopus oocytes. J Biol Chem, 2001. 276(49): p. 46523-32.Pubmed

12. Esposito, G., et al., Cellular and functional defects in a mouse model of heart failure. Am J Physiol Heart Circ Physiol, 2000. 279(6): p. H3101-12.Pubmed

13. Wessely, R., et al., Transgenic expression of replication-restricted enteroviral genomes in heart muscle induces defective excitation-contraction coupling and dilated cardiomyopathy. J Clin Invest, 1998. 102(7): p. 1444-53.Pubmed

14. Santana, L.F., A.M. Gomez, and W.J. Lederer, calcium flux through promiscuous cardiac sodium channels: slip-mode conductance. Science, 1998. 279(5353): p. 1027-33.Pubmed

15. Santana, L.F., E.G. Kranias, and W.J. Lederer, Calcium sparks and excitation-contraction coupling in phospholamban- deficient mouse ventricular myocytes. J Physiol, 1997. 503(Pt 1): p. 21-9.Pubmed

16. Santana, L.F., et al., Amount of calcium in the sarcoplasmic reticulum: influence on excitation-contraction coupling in heart muscle. Heart Vessels, 1997. Suppl(12): p. 44-9.Pubmed

17. Gomez, A.M., et al., Defective excitation-contraction coupling in experimental cardiac hypertrophy and heart failure. Science, 1997. 276(5313): p. 800-6.Pubmed

18. Santana, L.F., et al., Relation between the sarcolemmal calcium current and calcium sparks and local control theories for cardiac excitation-contraction coupling. Circ Res, 1996. 78(1): p. 166-71.Pubmed

19. Klein, M.G., et al., Two mechanisms of quantized calcium release in skeletal muscle. Nature, 1996. 379(6564): p. 455-8.Pubmed

20. Nelson, M.T., et al., Relaxation of arterial smooth muscle by calcium sparks. Science, 1995. 270(5236): p. 633-7.Pubmed